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    北京伯樂生命科學發展有限公司上海辦事處 (上海天崛電子科技有限公司)

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    北京伯樂生命科學發展有限公司上海辦事處 (上海天崛電子科技有限公司)>細胞株> 美國ATCC小鼠垂體瘤細胞株GT1.1分泌促生長激素分泌激素
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    美國ATCC小鼠垂體瘤細胞株GT1.1分泌促生長激素分泌激素

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    • 北京伯樂生命科學發展有限公司上海辦事處 (上海天崛電子科技有限公司)
    • 2018-02-17 13:21:38
    • 上海市
    • 美國
    • 6014

    我要詢價

    【簡單介紹】

    本公司長期經營各大細胞株。價格實惠,質量有保證。詳細價格請咨詢在線人員。全國統一訂購: 全國統一售后: 訂貨139_17814677

    【詳細說明】

    推薦培養基:(FM, Cat. No. 2301)

    儲存:液氮

    運輸:干冰

    用途:科研

    The most abundant cell type in lung interstitium is fibroblasts. They resemble ordinary fibroblasts but have some distinguishing features, for examle, they have long branching processes and gap junctions. Their principle function is production of type III collagen, elastin, and proteoglycans of the extracellular matrix of the alveolar septa. Pulmonary fibroblasts (PF) play an important role in the repair and remodeling processes following injury. The controlled accumulation of fibroblasts to sites of inflammation is crucial to effective tissue repair after injury [1]. Either inadequate or excessive accumulation of fibroblasts could result in abnormal tissue function. For examle, the excess proliferation of fibroblasts contributes to the adventitial thickening observed during the development of hypoxia-induced pulmonary hypertension [2].

    HPF from ScienCell Research Laboratories are isolated from human lung tissue. HPF are cryopreserved at primary culture and delivered frozen. Each vial contains >5 x 105 cells in 1 ml volume. HPF are characterized by immunofluorescent method with antibody to fibronectin. HPF are negative for HIV-1, HBV, HCV, mycoplasma, bacteria, yeast and fungi. HPF are guaranteed to further expand for 15 population doublings at the conditions provided by ScienCell Research Laboratories.

    Reference
    [1] Kuwano K, Hagimoto N, Hara N. (2001) Molecular mechanisms of pulmonary fibrosis and current treatment. Curr Mol Med 1(5):551-73.
    [2] Das M, Dempsey EC, Reeves JT, Stenmark KR. (2004) Selective expansion of fibroblast subpopulations from pulmonary artery adventitia in response to hypoxia. Am J Physiol Lung Cell Mol Physiol 282(5):L976-86

     

        
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